Difference between revisions of "Sudden natural death"
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Notes: | Notes: | ||
*The mechanism is usually [[cardiac arrhythmia|arrhythmia]]. | *The mechanism is usually [[cardiac arrhythmia|arrhythmia]]. | ||
**Generally, this is usually not provable at [[autopsy]]; however, some findings have been proposed. | **Generally, this is usually not provable at [[autopsy]]; however, some findings have been proposed.<ref name=pmid15837088>{{Cite journal | last1 = Baroldi | first1 = G. | last2 = Silver | first2 = MD. | last3 = Parolini | first3 = M. | last4 = Pomara | first4 = C. | last5 = Turillazzi | first5 = E. | last6 = Fineschi | first6 = V. | title = Myofiberbreak-up: a marker of ventricular fibrillation in sudden cardiac death. | journal = Int J Cardiol | volume = 100 | issue = 3 | pages = 435-41 | month = Apr | year = 2005 | doi = 10.1016/j.ijcard.2004.10.007 | PMID = 15837088 }}</ref> | ||
====Detailed cardiac<ref name=pmid16410164/>==== | ====Detailed cardiac<ref name=pmid16410164/>==== | ||
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**Sodium channel disease. | **Sodium channel disease. | ||
**Idiopathic ventricular fibrillation. | **Idiopathic ventricular fibrillation. | ||
*Tumours. | |||
**[[Cystic tumour of the atrioventricular nodal region]]. | |||
Post-mortem (molecular) testing for arrhythmias:<ref>MSP. 29 September 2010:</ref> | Post-mortem (molecular) testing for arrhythmias:<ref>MSP. 29 September 2010:</ref> | ||
| Line 55: | Line 57: | ||
*Ruptured AAA. | *Ruptured AAA. | ||
*Peptic ulcer. | *Peptic ulcer. | ||
*Cerebral aneurysm. | *[[Cerebral aneurysm]]. | ||
==See also== | ==See also== | ||
Latest revision as of 05:18, 21 July 2016
Sudden natural death happens. It must be differentiated from other ways of dying (suicide, homicide, accidental).
By system
Respiratory[1]
Cerebral
Sudden cardiac death
Older
- Atherosclerotic heart disease (ASHD); AKA coronary artery disease (CAD).
- Hypertensive heart disease - a heart > ~400 g is considered good enough if nothing else is present.[2]
Younger
- Arrhythmogenic right ventricular cardiomyopathy.
- Hypertrophic cardiomyopathy, e.g. hypertrophic obstructive cardiomyopathy (HOCM).
Notes:
- The mechanism is usually arrhythmia.
Detailed cardiac[1]
- ASHD.
- Cardiomyopathy.
- Lymphocytic myocarditis.
- Floppy mitral valve.
- Aortic valve stenosis.
- Congenital cardiac abnormality.
- Coronary artery dissection.
- Aortic dissection.
- Arrhythmia.[4]
- Long QT syndrome.
- Brugada syndrome.
- Short QT syndrome.
- Catecholaminergic polymorphic ventricular tachycardia (CPVT).
- Anomalous conduction pathways.
- Dysplasia of nodal arteries.
- Atrioventricular node tumour.
- Sodium channel disease.
- Idiopathic ventricular fibrillation.
- Tumours.
Post-mortem (molecular) testing for arrhythmias:[5]
- CPVT.
- Sodium channel disease.
- Brugada syndrome.
By mechanism
Hemorrhagic[1]
- Ruptured AAA.
- Peptic ulcer.
- Cerebral aneurysm.
See also
Reference
- ↑ 1.0 1.1 1.2 de la Grandmaison GL (January 2006). "Is there progress in the autopsy diagnosis of sudden unexpected death in adults?". Forensic Sci. Int. 156 (2-3): 138–44. doi:10.1016/j.forsciint.2004.12.024. PMID 16410164.
- ↑ MSP. 29 September 2010.
- ↑ Baroldi, G.; Silver, MD.; Parolini, M.; Pomara, C.; Turillazzi, E.; Fineschi, V. (Apr 2005). "Myofiberbreak-up: a marker of ventricular fibrillation in sudden cardiac death.". Int J Cardiol 100 (3): 435-41. doi:10.1016/j.ijcard.2004.10.007. PMID 15837088.
- ↑ URL: http://www.sads.org.uk/causes_of_sads.htm. Accessed on: 29 September 2010.
- ↑ MSP. 29 September 2010: