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==Porphyria cutanea tarda== | ==Porphyria cutanea tarda== | ||
=== | ===General=== | ||
Etiology: | |||
*Genetic, autosomal dominant. | *Genetic, autosomal dominant. | ||
Treatment: | |||
* | *D/C aggravating substances (see below) - phlebotomy, hydroxychloroquine if phlebotomy contraindicated. | ||
===Associations=== | Note: | ||
Medications/ | *Fits into a larger category of ''porphyria''. | ||
====Associations==== | |||
Medications/substances: | |||
*[[EtOH]], Rx (estrogen, [[NSAID]]s). | *[[EtOH]], Rx (estrogen, [[NSAID]]s). | ||
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*[[HIV]], [[hepatitis C]]. | *[[HIV]], [[hepatitis C]]. | ||
=== | ===Gross=== | ||
* | *In photoexposed areas subjected to trauma. | ||
===Microscopic=== | |||
Features:<ref>{{Ref PBoD8|1197}}</ref> | |||
*Subepidermal vesicles. | |||
*Thickening of superficial dermal blood vessels. | |||
Images: | |||
*[http://www.dermpedia.org/files/images/Image1_4.jpg Subepidermal blistering with thick vessels (dermpedia.org)]. | |||
==Epidermolysis bullosa== | ==Epidermolysis bullosa== |
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