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Difference between revisions of "Talk:Placenta"
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→Chorangiosis? Hypertrophic decidual vasculopathy?: new section
(Created page with "==Infarcts== ===Gross description=== The specimen consists of a singleton placenta, received fresh in a container with patient identifiers. Umbilical Cord: *No. of segments: one...") |
(→Chorangiosis? Hypertrophic decidual vasculopathy?: new section) |
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The extent of the villitis suggests this finding is probably inconsequential; however, it should be noted that severe lymphohistiocytic villitis is associated with adverse pregnancy outcomes, in a subset of patients. | The extent of the villitis suggests this finding is probably inconsequential; however, it should be noted that severe lymphohistiocytic villitis is associated with adverse pregnancy outcomes, in a subset of patients. | ||
== Chorangiosis? Hypertrophic decidual vasculopathy? == | |||
==Diagnosis== | |||
PLACENTA, BIRTH:<br> | |||
- THREE VESSEL CORD.<br> | |||
- SMALL SUBCHORIONIC HEMORRHAGE.<br> | |||
- NORMAL THIRD TRIMESTER VILLI AND VILLI WITH FEATURES SUGGESTIVE OF CHORANGIOSIS, SEE COMMENT 1.<br> | |||
- NEGATIVE FOR CHORIOAMNIONITIS AND NEGATIVE FOR MECONIUM.<br> | |||
- THICK-WALLED MATERNAL VESSELS (LACK OF PHYSIOLOGIC CONVERSION), SEE COMMENT 2. | |||
==Comment== | |||
Comment 1: | |||
Villi, focally, have increased vascular channels; however, they are the appropriate size, for the gestational age, and have peripheral vessels. | |||
Comment 2: | |||
Lack of physiologic conversion (of the spiral arteries of the uterus) is a feature seen in hypertrophic decidual vasculopathy (the histopathologic correlate of gestational hypertension). Other changes of hypertrophic decidual vasculopathy (atherosis, intimal hyperplasia, fibrinoid necrosis of the vessel wall, perivascular inflammation, thrombosis) are absent. | |||