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Lack of physiologic conversion (of the spiral arteries of the uterus) is a feature seen in hypertrophic decidual vasculopathy (the histopathologic correlate of gestational hypertension). Other changes of hypertrophic decidual vasculopathy (atherosis, intimal hyperplasia, fibrinoid necrosis of the vessel wall, perivascular inflammation, thrombosis) are absent. | Lack of physiologic conversion (of the spiral arteries of the uterus) is a feature seen in hypertrophic decidual vasculopathy (the histopathologic correlate of gestational hypertension). Other changes of hypertrophic decidual vasculopathy (atherosis, intimal hyperplasia, fibrinoid necrosis of the vessel wall, perivascular inflammation, thrombosis) are absent. | ||
== Decidual inflammation == | |||
<pre> | |||
PLACENTA, UMBILICAL CORD AND FETAL MEMBRANES, BIRTH: | |||
- FETAL MEMBRANES WITH MILD DECIDUAL INFLAMMATION (LYMPHOCYTES AND NEUTROPHILS), | |||
OTHERWISE WITHIN NORMAL LIMITS. | |||
- PLACENTAL DISC WITH THIRD TRIMESTER VILLI, MILD PERIVILLOUS FIBRIN DEPOSITION AND | |||
SUBCHORIONIC FIBRIN (1 CM MAXIMAL DIMENSION). | |||
- THREE VESSEL UMBILICAL CORD WITHIN NORMAL LIMITS. | |||
</pre> |
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